Atrial tachycardia secondary to sino-atrial node reentry.
نویسندگان
چکیده
In one patient reprodncibk episodes of sMal tachyear(200 msec, control; 70 msec, atropine) and sustained dia conld be elicited via programmed atrial premature atrial tachycardia no longer could be elicited. These dcpoluizations. Evllodion of the elechpphysiologic studies mpport the concept that h a t t i a l reciprocation shldks suggested that the most tikely mechanism was can be a mechanism responsible for atrial tacharrhythdm-athd miprocation. Intmvmom atropine prodnced mins in mau. a pronomced shortening of do-atrial conduction time
منابع مشابه
تأثیــر نیکــل بر مسدود کردن جریان یونــی گذرای کلسیمــی سلولهای گــره سینوسی ـ دهلیزی دست نخورده و سالم قلب خرگوش
The mammalian sino-atrial node is not a uniform tissue in histological and electrophysiological terms. The differences in ionic currents underlying the regional differences in electrial activity are only just beginning to be understood. One of the ionic currents it is thought to play a role in the center of sino-atrial node for action potential upstroke should be transient Ca+2 current ...
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عنوان ژورنال:
- Chest
دوره 66 5 شماره
صفحات -
تاریخ انتشار 1974